hunting journal
hunting journal
Here is a simple analogy to humans and foods. In the distant past, humans hunted for their food. It was generally a period feast or starve. When times are good, humans ate well. When hunting became scarce, people are starving. The body developed "thrifty gene" to convert energy into fat and store fat for future use. She has also developed enzymes and peptides that controlled our appetites. In simple terms, the body finally came in the way of celebration or die of hunger and increased our appetites when we lost weight.
Factors affecting the stronger are weight gain genetics, race and ethnicity and age. The size of the human body has a genetic component important. Some 70% of body weight a person is genetically determined and about 30% are influenced by the environment. Having parents obese doubles the risk for their children to be obese at some point in their lives. There are about 400 genes responsible for determining the weight. Genes contribute to obesity by acting on appetite, our feeling of fullness, metabolism, food cravings, body-fat distribution, and the tendency to use food as a way of coping with stress. The journal Science reported in 2006 that people have two copies of a variant specific gene (INSIG-2) were 22% more likely to have a BMI over 30. It is believed that INSIG-2 affects the regulation of another gene involved in fat production. Being genetically predisposed to gaining weight does not automatically mean that you will become obese. Research suggests that for some people, genes account for only 25% of the predisposition to overweight, while for others the influence Genetics is as high as 70% to 80%. Your eating habits and level of physical activity are important factors in the amount of weight you win.
Studies have shown that a peptide called leptin may contribute an important piece to obesity. We all a set of "point" with respect to the weight. Our bodies like to maintain weight in a specific range. Leptin is part of the mechanism set point. If you reduce your body weight by 10%, leptin is reduced by almost 53%. This turns on NPY in the hypothalamus, which increases your urge to eat. If you increase your body weight by 10%, leptin increases of 300%, turn off your desire to eat. Postmenopausal women have less leptin than women premenopausal and NPY, making them want to eat more. Some people have heavy fixed high levels of leptin, but they have developed a insensitivity to this if they desire to eat continues unabated.
In the 1990s, there was an investigation of the gene coding for the lipoprotein lipase (LPL), an enzyme produced by fat cells to store calories as fat. LPL is partly controlled by estrogen in women and testosterone in men. In women, the fat cells in hips, thighs and breasts secrete LPL. In men, LPL is produced by fat cells in the stomach region. Fat cells in the region abdominal release their contents for quick energy, while fat in the thighs and buttocks are used to store energy in the long term. This means that is easier for a man to lose his belly for a woman to lose weight in her thighs and buttocks. LPL also makes it easier to regain weight. The researchers found that levels of LPL increased after weight loss. They believe that weight loss activated the gene producing the enzyme. This May be one reason why it is easier for a dieter to regain lost weight for someone who has never been obese to put on weight.
Wayne Mcgregor has been on a degree course in nutrition and dietetics, and has a diploma in fitness training, as well as a wealth of experience in helping people to lose weight and build muscle. His website provides hundreds of free articles on losing weight and includes tools, sample diets, and information on different foods.
http://www.weightlossforall.com
Mikes Hunting Journal
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